Tumor necrosis factor alpha inhibits wound healing in the rat.

Tumor necrosis factor and wound healing.

$Multiple roles of tumor necrosis factor-alpha in fracture healing.$
Multiple roles of tumor necrosis factor-alpha in fracture healing.

Tumor necrosis factor alpha inhibits hepatocyte mitochondrial respiration.

Tumor necrosis factor-alpha inhibits human chorionic gonadotropin secretion.

Tumor necrosis factor-alpha inhibits expression of pulmonary surfactant protein.

Tumor necrosis factor alpha inhibits ovulation and induces granulosa cell death in rat ovaries.

Tumor necrosis factor-alpha.

tumor necrosis factor on wound healing.

Tumor necrosis factor alpha in mycobacterial infection.

Tumor necrosis factor-alpha in human milk.

Wound healing potential of pterospermum acerifolium wild. With induction of tumor necrosis factor - α.

Tumor necrosis factor alpha inhibits the hormonal response of the pituitary gland to hypothalamic releasing factors.

Cytotoxic mechanism of tumor necrosis factor-alpha.

Tumor necrosis factor alpha inhibits gonadotropin action in cultured porcine Leydig cells: site(s) of action.

Tumor necrosis factor-alpha inhibits albumin gene expression in a murine model of cachexia.

The effects of tumor necrosis factor-alpha on systolic and diastolic function in rat ventricular myocytes.

A healing promoting factor in rat wound fluid.

Increased expression of tumor necrosis factor-alpha in the rat hippocampus after acute homocysteine administration.

Histopathological effects of intracerebral injections of human recombinant tumor necrosis factor-alpha in the rat.

Interactions between tumor necrosis factor-alpha, hypothalamic corticotropin-releasing hormone, and adrenocorticotropin secretion in the rat.

Interleukin-1 alpha inhibits the effects of gamma-interferon and tumor necrosis factor alpha on the expression of the major histocompatibility antigens by the rat endothelium.

Thalidomide selectively inhibits tumor necrosis factor alpha production by stimulated human monocytes.

The molecular action of tumor necrosis factor-alpha.

Dual role of tumor necrosis factor-alpha in angiogenesis.

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Tumor necrosis factor and wound healing.

Multiple roles of tumor necrosis factor-alpha in fracture healing.

Tumor necrosis factor alpha inhibits hepatocyte mitochondrial respiration.

Tumor necrosis factor-alpha inhibits human chorionic gonadotropin secretion.

Tumor necrosis factor-alpha inhibits expression of pulmonary surfactant protein.

Tumor necrosis factor alpha inhibits ovulation and induces granulosa cell death in rat ovaries.

Tumor necrosis factor-alpha.

tumor necrosis factor on wound healing.

Tumor necrosis factor alpha in mycobacterial infection.

Tumor necrosis factor-alpha in human milk.

Wound healing potential of pterospermum acerifolium wild. With induction of tumor necrosis factor - α.

Tumor necrosis factor alpha inhibits the hormonal response of the pituitary gland to hypothalamic releasing factors.

Cytotoxic mechanism of tumor necrosis factor-alpha.

Tumor necrosis factor alpha inhibits gonadotropin action in cultured porcine Leydig cells: site(s) of action.

Tumor necrosis factor-alpha inhibits albumin gene expression in a murine model of cachexia.

The effects of tumor necrosis factor-alpha on systolic and diastolic function in rat ventricular myocytes.

A healing promoting factor in rat wound fluid.

Increased expression of tumor necrosis factor-alpha in the rat hippocampus after acute homocysteine administration.

Histopathological effects of intracerebral injections of human recombinant tumor necrosis factor-alpha in the rat.

Interactions between tumor necrosis factor-alpha, hypothalamic corticotropin-releasing hormone, and adrenocorticotropin secretion in the rat.

Interleukin-1 alpha inhibits the effects of gamma-interferon and tumor necrosis factor alpha on the expression of the major histocompatibility antigens by the rat endothelium.

Thalidomide selectively inhibits tumor necrosis factor alpha production by stimulated human monocytes.

The molecular action of tumor necrosis factor-alpha.

Dual role of tumor necrosis factor-alpha in angiogenesis.

Tumor necrosis factor alpha inhibits wound healing in the rat.

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