Normal development of nerve-muscle synapses in mice lacking the prion protein gene.

Normal development and behaviour of mice lacking the neuronal cell-surface PrP protein.

Prion protein scrapie and the normal cellular prion protein.

The sequential development of abnormal prion protein accumulation in mice with Creutzfeldt-Jakob disease.

Unusual cerebral vascular prion protein amyloid distribution in scrapie-infected transgenic mice expressing anchorless prion protein.

Analysis of the prion protein gene in multiple system atrophy.

Codon 129 changes in the prion protein gene in Caucasians.

Sialic Acid within the Glycosylphosphatidylinositol Anchor Targets the Cellular Prion Protein to Synapses.

Deletion in the prion protein gene in a demented patient.

BSE-associated polymorphisms in the prion protein gene: an investigation.

Aberrant Cerebellar Development in Mice Lacking Dual Oxidase Maturation Factors.

Cellular prion protein is present in mitochondria of healthy mice.

Genomic structure of the human prion protein gene.

Normal development and function of CD8+ cells but markedly decreased helper cell activity in mice lacking CD4.

Hyperammonemia in gene-targeted mice lacking functional hepatic glutamine synthetase.

Chimeric prion protein expression in cultured cells and transgenic mice.

Spontaneous neurodegeneration in transgenic mice with mutant prion protein.

Development of Th17-Associated Interstitial Kidney Inflammation in Lupus-Prone Mice Lacking the Gene Encoding STAT-1.

Age-dependent neuromuscular impairment in prion protein knockout mice.

EBP homologous protein CHOP.

Survival of functional synapses on crustacean neurons lacking cell bodies.

Normal cerebellar development in S100B-deficient mice.

Review of studies that have used knockout mice to assess normal function of prion protein under immunological or pathophysiological stress.

Prion protein participates in the protection of mice from lipopolysaccharide infection by regulating the inflammatory process.

Adult mice lacking the type 2 iodothyronine deiodinase have increased subchondral bone but normal articular cartilage.

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Normal development and behaviour of mice lacking the neuronal cell-surface PrP protein.

Prion protein scrapie and the normal cellular prion protein.

The sequential development of abnormal prion protein accumulation in mice with Creutzfeldt-Jakob disease.

Unusual cerebral vascular prion protein amyloid distribution in scrapie-infected transgenic mice expressing anchorless prion protein.

Analysis of the prion protein gene in multiple system atrophy.

Codon 129 changes in the prion protein gene in Caucasians.

Sialic Acid within the Glycosylphosphatidylinositol Anchor Targets the Cellular Prion Protein to Synapses.

Deletion in the prion protein gene in a demented patient.

BSE-associated polymorphisms in the prion protein gene: an investigation.

Aberrant Cerebellar Development in Mice Lacking Dual Oxidase Maturation Factors.

Cellular prion protein is present in mitochondria of healthy mice.

Genomic structure of the human prion protein gene.

Normal development and function of CD8+ cells but markedly decreased helper cell activity in mice lacking CD4.

Hyperammonemia in gene-targeted mice lacking functional hepatic glutamine synthetase.

Chimeric prion protein expression in cultured cells and transgenic mice.

Spontaneous neurodegeneration in transgenic mice with mutant prion protein.

Development of Th17-Associated Interstitial Kidney Inflammation in Lupus-Prone Mice Lacking the Gene Encoding STAT-1.

Age-dependent neuromuscular impairment in prion protein knockout mice.

EBP homologous protein CHOP.

Survival of functional synapses on crustacean neurons lacking cell bodies.

Normal cerebellar development in S100B-deficient mice.

Review of studies that have used knockout mice to assess normal function of prion protein under immunological or pathophysiological stress.

Prion protein participates in the protection of mice from lipopolysaccharide infection by regulating the inflammatory process.

Adult mice lacking the type 2 iodothyronine deiodinase have increased subchondral bone but normal articular cartilage.

Normal development of nerve-muscle synapses in mice lacking the prion protein gene.

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