the majority of cases, but a complete gland removal certainly can expose the patient to such complication. The accessory glands cannot be counted on
to
keep the eye moist at all times. Claes H. Boston
Flow Sheet for the of the Fluorescein
Dohlman, MD
Interpretation Angiograms
To the
Editor.\p=m-\Tworecent academy (1974) in which I was involved utilized an original teaching tool called a "flow sheet," representing a logical thought process for the interpretation of fluorescein angiograms. courses
One of the courses pertained to the macula and was given along with J. Donald M. Gass and A. Edward Maumenee. The topic of the other course was fluorescein angiographic interpretation, and it was given with Thomas Burton, Maurice Rabb, and Lawrence Yannuzzi. Flow sheet for
interpretation
of fluorescein
Changes in Neuronal Ceroid-Lipofuscinosis To the Editor.\p=m-\Thearticle entitled
"Ceroid-Lipofuscinosis (Batten Disease)" by Hittner and Zeller that appeared in the March issue of the Ar-
Our experience at these courses demonstrated that the flow sheet was a valuable method for teaching the basics of understanding and using the fluorescein angiogram as an in vivo approach to gaining knowledge of posterior polar histopathology. Currently, Drs Burton, Rabb, Yannuzzi, and I are developing the flow sheet as part of a textbook and atlas to be published some time next year. The original version of the flow sheet used at the Academy meeting in the fall of 1974 is presented here (Figure). Howard Schatz, MD San Francisco angiogram.
ABNORMAL FLUORESCENCE CYSTOID EDEMA SENSORY DETACHMENT RPE DETACHMENT
NON-CYSTOID EDEMA
DRUSEN SCAR
tional, and four
LAMINA CRIBROSA TRANSMISSION —
PIGMENT EPITHEL IAL WINDOW DEFECT
of NCL. The chronic type is perhaps of greatest interest to ophthalmolo¬ gists since visual symptoms are frequently the major reason such pa¬ tients seek medical attention. Deteri¬ oration in mental and motor capabili¬ ties is slow and is frequently outweighed by the severe visual im¬ pairment. Routine funduscopic exam¬ ination shows very typical retinal findings that are a composite of (1) a granular macular deterioration asso¬ ciated with severe impairment of cen¬ tral vision; (2) patchy retinal atrophy that probably accounts for the severe attenuation of retinal vessels; and (3) optic atrophy of the so-called waxy yellow type. In a rare instance, one sees peripheral pigmentation and de¬ pigmentation, but this, in our experi¬ ence, has never been of the severity or character of typical retinitis pig¬
< DRUSEN TORTUOSITY. DILATATION. ETC. NEOVASCULARIZATION MICRO MACRO TELANGIECTASIAS SHUNTS, COLLATERALS ANEURYSMS-
V_ ABNORMAL < VESSELS
NEOVASCULARIZATION VESSELS IN SCAR .,.„,„
RETINAL
____-ANGIOMAS
to
keep the eye moist at all times. Claes H. Boston
Flow Sheet for the of the Fluorescein
Dohlman, MD
Interpretation Angiograms
To the
Editor.\p=m-\Tworecent academy (1974) in which I was involved utilized an original teaching tool called a "flow sheet," representing a logical thought process for the interpretation of fluorescein angiograms. courses
One of the courses pertained to the macula and was given along with J. Donald M. Gass and A. Edward Maumenee. The topic of the other course was fluorescein angiographic interpretation, and it was given with Thomas Burton, Maurice Rabb, and Lawrence Yannuzzi. Flow sheet for
interpretation
of fluorescein
Changes in Neuronal Ceroid-Lipofuscinosis To the Editor.\p=m-\Thearticle entitled
"Ceroid-Lipofuscinosis (Batten Disease)" by Hittner and Zeller that appeared in the March issue of the Ar-
Our experience at these courses demonstrated that the flow sheet was a valuable method for teaching the basics of understanding and using the fluorescein angiogram as an in vivo approach to gaining knowledge of posterior polar histopathology. Currently, Drs Burton, Rabb, Yannuzzi, and I are developing the flow sheet as part of a textbook and atlas to be published some time next year. The original version of the flow sheet used at the Academy meeting in the fall of 1974 is presented here (Figure). Howard Schatz, MD San Francisco angiogram.
ABNORMAL FLUORESCENCE CYSTOID EDEMA SENSORY DETACHMENT RPE DETACHMENT
NON-CYSTOID EDEMA
DRUSEN SCAR
tional, and four
LAMINA CRIBROSA TRANSMISSION —
PIGMENT EPITHEL IAL WINDOW DEFECT
of NCL. The chronic type is perhaps of greatest interest to ophthalmolo¬ gists since visual symptoms are frequently the major reason such pa¬ tients seek medical attention. Deteri¬ oration in mental and motor capabili¬ ties is slow and is frequently outweighed by the severe visual im¬ pairment. Routine funduscopic exam¬ ination shows very typical retinal findings that are a composite of (1) a granular macular deterioration asso¬ ciated with severe impairment of cen¬ tral vision; (2) patchy retinal atrophy that probably accounts for the severe attenuation of retinal vessels; and (3) optic atrophy of the so-called waxy yellow type. In a rare instance, one sees peripheral pigmentation and de¬ pigmentation, but this, in our experi¬ ence, has never been of the severity or character of typical retinitis pig¬
< DRUSEN TORTUOSITY. DILATATION. ETC. NEOVASCULARIZATION MICRO MACRO TELANGIECTASIAS SHUNTS, COLLATERALS ANEURYSMS-
V_ ABNORMAL < VESSELS
NEOVASCULARIZATION VESSELS IN SCAR .,.„,„
RETINAL
____-ANGIOMAS
