Frequency of ultraviolet light-induced mutations is higher in xeroderma pigmentosum variant cells than in normal human cells.

Ultraviolet mutagenesis of normal and xeroderma pigmentosum variant human fibroblasts.

Similar distributions of repaired sites in chromatin of normal and xeroderma pigmentosum variant cells damaged by ultraviolet light.

Correction of the ultraviolet light induced DNA-repair defect in xeroderma pigmentosum cells by electroporation of a normal human endonuclease.

Reactivation of psoralen-reacted plasmid DNA in Fanconi anemia, xeroderma pigmentosum, and normal human fibroblast cells.

DNA-protein cross-linking by ultraviolet radiation in normal human and xeroderma pigmentosum fibroblasts.

Defective recovery of semi-conservative DNA synthesis in xeroderma pigmentosum cells following split-dose ultraviolet irradiation.

Ultraviolet mutational spectrum in a shuttle vector propagated in xeroderma pigmentosum lymphoblastoid cells and fibroblasts.

Ultraviolet light induction of diphtheria toxin-resistant mutants of normal and xeroderma pigmentosum human fibroblasts.

Xeroderma pigmentosum variant cells are less likely than normal cells to incorporate dAMP opposite photoproducts during replication of UV-irradiated plasmids.

Induction of sister chromatid exchanges in xeroderma pigmentosum cells after exposure to ultraviolet light.

Electroporation of normal human DNA endonucleases into xeroderma pigmentosum cells corrects their DNA repair defect.

Molecular analysis of ultraviolet-induced mutations in a xeroderma pigmentosum cell line.

Photoreactivation of pyrimidine dimers in the DNA of normal and xeroderma pigmentosum cells.

Phage T4 endonuclease V stimulates DNA repair replication in isolated nuclei from ultraviolet-irradiated human cells, including xeroderma pigmentosum fibroblasts.

High frequency of PTEN mutations in nevi and melanomas from xeroderma pigmentosum patients.

Mutational hotspot variability in an ultraviolet-treated shuttle vector plasmid propagated in xeroderma pigmentosum and normal human lymphoblasts and fibroblasts.

Recovery of DNA synthesis after ultraviolet irradiation of xeroderma pigmentosum cells depends on excision repair and is blocked by caffeine.

Xeroderma pigmentosum cells contain low levels of photoreactivating enzyme.

Xeroderma pigmentosum.

Carrier detection in xeroderma pigmentosum.

Delayed hypersensitivity in xeroderma pigmentosum.

Xeroderma pigmentosum.

genotype in a cohort of 23 xeroderma pigmentosum-variant patients reveals 12 new disease-causing POLH mutations.

Cellular responses to hematoporphyrin-induced photooxidative damage in Fanconi anemia, xeroderma pigmentosum and normal human fibroblasts.

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Ultraviolet mutagenesis of normal and xeroderma pigmentosum variant human fibroblasts.

Similar distributions of repaired sites in chromatin of normal and xeroderma pigmentosum variant cells damaged by ultraviolet light.

Correction of the ultraviolet light induced DNA-repair defect in xeroderma pigmentosum cells by electroporation of a normal human endonuclease.

Reactivation of psoralen-reacted plasmid DNA in Fanconi anemia, xeroderma pigmentosum, and normal human fibroblast cells.

DNA-protein cross-linking by ultraviolet radiation in normal human and xeroderma pigmentosum fibroblasts.

Defective recovery of semi-conservative DNA synthesis in xeroderma pigmentosum cells following split-dose ultraviolet irradiation.

Ultraviolet mutational spectrum in a shuttle vector propagated in xeroderma pigmentosum lymphoblastoid cells and fibroblasts.

Ultraviolet light induction of diphtheria toxin-resistant mutants of normal and xeroderma pigmentosum human fibroblasts.

Xeroderma pigmentosum variant cells are less likely than normal cells to incorporate dAMP opposite photoproducts during replication of UV-irradiated plasmids.

Induction of sister chromatid exchanges in xeroderma pigmentosum cells after exposure to ultraviolet light.

Electroporation of normal human DNA endonucleases into xeroderma pigmentosum cells corrects their DNA repair defect.

Molecular analysis of ultraviolet-induced mutations in a xeroderma pigmentosum cell line.

Photoreactivation of pyrimidine dimers in the DNA of normal and xeroderma pigmentosum cells.

Phage T4 endonuclease V stimulates DNA repair replication in isolated nuclei from ultraviolet-irradiated human cells, including xeroderma pigmentosum fibroblasts.

High frequency of PTEN mutations in nevi and melanomas from xeroderma pigmentosum patients.

Mutational hotspot variability in an ultraviolet-treated shuttle vector plasmid propagated in xeroderma pigmentosum and normal human lymphoblasts and fibroblasts.

Recovery of DNA synthesis after ultraviolet irradiation of xeroderma pigmentosum cells depends on excision repair and is blocked by caffeine.

Xeroderma pigmentosum cells contain low levels of photoreactivating enzyme.

Xeroderma pigmentosum.

Carrier detection in xeroderma pigmentosum.

Delayed hypersensitivity in xeroderma pigmentosum.

Xeroderma pigmentosum.

genotype in a cohort of 23 xeroderma pigmentosum-variant patients reveals 12 new disease-causing POLH mutations.

Cellular responses to hematoporphyrin-induced photooxidative damage in Fanconi anemia, xeroderma pigmentosum and normal human fibroblasts.

Frequency of ultraviolet light-induced mutations is higher in xeroderma pigmentosum variant cells than in normal human cells.

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