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Cell killing by viruses. II. Cell killing by vesicular stomatitis virus: a requirement for virion-derived transcription.
Cell killing by viruses. V. Transcribing defective interfering particles of vesicular stomatitis virus function as cell-killing particles.
The specific inhibition of vesicular stomatitis virus replication by toyocamycin.
Cell killing by frog virus 3: evidence for cell killing by single viral particles or single viral subunits.
Cell killing by Simian virus 40: protective effect of chloroquine.
Interferon action III. The rate of primary transcription of vesicular stomatitis virus is inhibited by interferon action.
Tumor cell killing by macrophages activated in vitro with lymphocyte mediators. III. Inhibition by cytochalasins, colchicine, and vinblastine.
Inhibition of transcription by immunoglobulins directed against the ribonucleoprotein of homotypic and heterotypic vesicular stomatitis viruses.
Enveloped viruses as model membrane systems: microviscosity of vesicular stomatitis virus and host cell membranes.
Rescue of vesicular stomatitis virus from homologous and heterologous interferon-induced resistance in human cell cultures by poxviruses.
Interferon induction by vesicular stomatitis virus and its role in virus replication.
Mechanisms of cancer cell killing by the adenovirus E4orf4 protein.
The inhibition of vesicular stomatitis virus protein synthesis by frog virus 3.
Vesicular stomatitis virus-based vaccines against Lassa and Ebola viruses.
Ganciclovir.
Inhibition of pseudorabies virus replication by vesicular stomatitis virus. II Activity of defective interfering particles.
Interactions of vesicular stomatitis virus with murine cell surface antigens.
Inhibition of vesicular stomatitis virus replication by frog virus 3. Selective action on secondary transcription.
Preferential generation of monoclonal IgG-producing hybridomas by use of vesicular stomatitis virus-mediated cell fusion.
Effect of certain inhibitors of glycoprotein synthesis on cell fusion induced by vesicular stomatitis virus.
A vesicular stomatitis virus glycoprotein epitope-incorporated oncolytic adenovirus overcomes CAR-dependency and shows markedly enhanced cancer cell killing and suppression of tumor growth.
RNA synthesis of vesicular stomatitis virus. VII. Complete separation of the mRNA's of vesicular stomatitis virus by duplex formation.
Cell killing by simian virus 40: impairment of membrane formation and function.
Pseudotypes of vesicular stomatitis virus with the mixed coat of reticuloendotheliosis virus and vesicular stomatitis virus.
Cell killing by viruses. III. The interferon system and inhibition of cell killing by vesicular stomatitis virus.
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Cell killing by viruses. II. Cell killing by vesicular stomatitis virus: a requirement for virion-derived transcription.
Cell killing by viruses. V. Transcribing defective interfering particles of vesicular stomatitis virus function as cell-killing particles.
The specific inhibition of vesicular stomatitis virus replication by toyocamycin.
Cell killing by frog virus 3: evidence for cell killing by single viral particles or single viral subunits.
Cell killing by Simian virus 40: protective effect of chloroquine.
Interferon action III. The rate of primary transcription of vesicular stomatitis virus is inhibited by interferon action.
Tumor cell killing by macrophages activated in vitro with lymphocyte mediators. III. Inhibition by cytochalasins, colchicine, and vinblastine.
Inhibition of transcription by immunoglobulins directed against the ribonucleoprotein of homotypic and heterotypic vesicular stomatitis viruses.
Enveloped viruses as model membrane systems: microviscosity of vesicular stomatitis virus and host cell membranes.
Rescue of vesicular stomatitis virus from homologous and heterologous interferon-induced resistance in human cell cultures by poxviruses.
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