Calcium activation of hyperpolarization response to acetylcholine in coronary endothelial cells.

Pharmacological distinction of the hyperpolarization response to caffeine and acetylcholine in guinea-pig coronary endothelial cells.

Characterization of acetylcholine-induced membrane hyperpolarization in endothelial cells.

Energetic response of coronary endothelial cells to hypoxia.

Activation of endothelial transient receptor potential C3 channel is required for small conductance calcium-activated potassium channel activation and sustained endothelial hyperpolarization and vasodilation of cerebral artery.

Electrical response of glioma cells to acetylcholine.

Coronary vasomotor response to acetylcholine relates to risk factors for coronary artery disease.

The calcium action potential and a prolonged calcium dependent after-hyperpolarization in mouse neuroblastoma cells.

Cytosolic calcium increase in coronary endothelial cells after H2O2 exposure and the inhibitory effect of U78517F.

Endothelial cells cultured from human umbilical vein release ATP, substance P and acetylcholine in response to increased flow.

Different sensitivities of acetylcholine-induced "after-hyperpolarization" compared to dopamine-induced hyperpolarization, to oubain or to lithium-replacement of sodium, in rabbit sympathetic ganglia.

Calcium mobilization by cadmium or decreasing extracellular Na+ or pH in coronary endothelial cells.

Endothelium-dependent relaxation to acetylcholine in the rabbit basilar artery: importance of membrane hyperpolarization.

The effect of external calcium and magnesium ions on the response of denervated muscle to acetylcholine.

PP168. The role of calcium supplementation in prevention endothelial cell activation, and possible relevance to preeclampsia.

Stimulation of phosphatidic acid synthesis in bovine aortic endothelial cells in response to activation of P2-purinergic receptors.

Selective coupling of different muscarinic acetylcholine receptors to neuronal calcium currents in DNA-transfected cells.

Acetylcholine released by endothelial cells facilitates flow-mediated dilatation.

Local and Circulating Endothelial Cells Undergo Endothelial to Mesenchymal Transition (EndMT) in Response to Musculoskeletal Injury.

Response of angiographically normal and atherosclerotic left anterior descending coronary arteries to acetylcholine.

Convective mass transfer effects on the intracellular calcium response of endothelial cells.

Activation of eNOS in endothelial cells exposed to ionizing radiation involves components of the DNA damage response pathway.

Mesenchymal Stem Cells Loaded with p5, Derived from CDK5 Activator p35, Inhibit Calcium-Induced CDK5 Activation in Endothelial Cells.

Activation of phospholipase C by the agonist U46619 is inhibited by cromakalim-induced hyperpolarization in porcine coronary artery.

Extracellular calcium participates in responses to acetylcholine in Xenopus oocytes.

Suggest Documents

Pharmacological distinction of the hyperpolarization response to caffeine and acetylcholine in guinea-pig coronary endothelial cells.

Characterization of acetylcholine-induced membrane hyperpolarization in endothelial cells.

Energetic response of coronary endothelial cells to hypoxia.

Activation of endothelial transient receptor potential C3 channel is required for small conductance calcium-activated potassium channel activation and sustained endothelial hyperpolarization and vasodilation of cerebral artery.

Electrical response of glioma cells to acetylcholine.

Coronary vasomotor response to acetylcholine relates to risk factors for coronary artery disease.

The calcium action potential and a prolonged calcium dependent after-hyperpolarization in mouse neuroblastoma cells.

Cytosolic calcium increase in coronary endothelial cells after H2O2 exposure and the inhibitory effect of U78517F.

Endothelial cells cultured from human umbilical vein release ATP, substance P and acetylcholine in response to increased flow.

Different sensitivities of acetylcholine-induced "after-hyperpolarization" compared to dopamine-induced hyperpolarization, to oubain or to lithium-replacement of sodium, in rabbit sympathetic ganglia.

Calcium mobilization by cadmium or decreasing extracellular Na+ or pH in coronary endothelial cells.

Endothelium-dependent relaxation to acetylcholine in the rabbit basilar artery: importance of membrane hyperpolarization.

The effect of external calcium and magnesium ions on the response of denervated muscle to acetylcholine.

PP168. The role of calcium supplementation in prevention endothelial cell activation, and possible relevance to preeclampsia.

Stimulation of phosphatidic acid synthesis in bovine aortic endothelial cells in response to activation of P2-purinergic receptors.

Selective coupling of different muscarinic acetylcholine receptors to neuronal calcium currents in DNA-transfected cells.

Acetylcholine released by endothelial cells facilitates flow-mediated dilatation.

Local and Circulating Endothelial Cells Undergo Endothelial to Mesenchymal Transition (EndMT) in Response to Musculoskeletal Injury.

Response of angiographically normal and atherosclerotic left anterior descending coronary arteries to acetylcholine.

Convective mass transfer effects on the intracellular calcium response of endothelial cells.

Activation of eNOS in endothelial cells exposed to ionizing radiation involves components of the DNA damage response pathway.

Mesenchymal Stem Cells Loaded with p5, Derived from CDK5 Activator p35, Inhibit Calcium-Induced CDK5 Activation in Endothelial Cells.

Activation of phospholipase C by the agonist U46619 is inhibited by cromakalim-induced hyperpolarization in porcine coronary artery.

Extracellular calcium participates in responses to acetylcholine in Xenopus oocytes.

Calcium activation of hyperpolarization response to acetylcholine in coronary endothelial cells.

Recommend Documents

Follow Us